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January 2009 - Volume 3, Issue 01

 

In this issue...

- Is Alzheimer disease caused by copper deficiency?

- Low vitamin D is linked to insulin resistance

- Selenium inversely associated with anemia

- Carnitine reduces oxidized LDL and triglycerides

- Clinical trial shows B vitamins affect autism

Animal experiments support theory that copper status affects Alzheimer's and decreased cognition is associated with low copper status in humans.

(Medical Hypothesis, 2008)

Link to ABSTRACT: Alzheimer Disease as copper deficiency

 

 

 

CLINICAL UPDATE - Low vitamin D is linked to insulin resistance

Recent studies suggest vitamin D is an important determinant for type 2 diabetes, and that genetics plays a major role in vitamin D levels throughout the body

(Journal of Nutrition, February 2009)

(American Journal of Clinical Nutrition, February 2009)

Link to ABSTRACT: Plasma 25-hydroxyvitamin D is associated with markers of the insulin resistant phenotype in nondiabetic adults.

   Link to ABSTRACT:  Genetic polymorphisms of the vitamin D binding protein and plasma concentrations of 25-hydroxyvitamin D in premenopausal women.

 

 

CLINICAL UPDATE - selenium inversely associated with anemia

Review of over 2000 adults aged 65 or older showed that those with the lowest selenium levels were almost three times more likely to have anemia.

(European Journal of Clinical Nutrition, January 2009)

Link to ABSTRACT: Low serum selenium is associated with anemia among older adults in the United States.

 

 

 

CLINICAL UPDATE - Carnitine reduces oxidized ldl and triglycerides

After three months of supplementation with L-carnitine, diabetic patients showed improvements in oxidized LDL levels, triglycerides and markers of oxidative stress.

(American Journal of Clinical Nutrition, January 2009)

Link to ABSTRACT: L-Carnitine supplementation reduces oxidized LDL cholesterol in patients with diabetes.

 

 

 

CLINICAL UPDATE - clinical trial shows b vitamins affect autism

Forty autistic children were treated with methylcobalamin and folinic acid for three months and demonstrated significant increases in concentrations of powerful antioxidants cysteine and glutathione during this period, as well as improvements in metabolite concentrations associated with autism.

(American Journal of Clinical Nutrition, January 2009)

Link to ABSTRACT: Efficacy of methylcobalamin and folinic acid treatment on glutathione redox status in children with autism.

 

 

 

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